Phase 1/1b Study of the Safety, Pharmacokinetics, Pharmacodynamics and Preliminary Clinical Activity of RP-6306 Alone or in Combination With RP-3500 or Debio 0123 in Patients With Advanced Solid Tumors

Study Identifier:
Debio 0123-106
CT.gov Identifier:
EudraCT Identifier:
EU Trial (CTIS) Number:
Study Contact Information:
N/A
Recruiting

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Study Summary

To evaluate RP-6306 in patients with cancer. To evaluate the assessment of safety, tolerability, dose and schedule (including the recommended Phase 2 dose). To assess the safety and tolerability of RP-6306 alone and in combination with RP-3500 or in combination with Debio 0123 in patients with eligible advanced solid tumors, determine the maximum tolerated dose (MTD) and assess preliminary anti-tumor activity. Evaluate the safety profile and MTD of RP-6306 alone and in combination with RP-3500 or in combination with Debio 0123 when administered orally to establish the recommended Phase 2 dose and schedule Characterize the PK and pharmacodynamics of RP-6306 alone and in combination with RP- 3500 or in combination with Debio 0123 Assess preliminary anti-tumor activity associated with RP-6306 alone and in combination with RP- 3500 or in combination with Debio 0123 Endpoints: safety, tolerability, RP2D, preliminary efficacy (RECIST v1.1, GCIG CA-125), clinical benefit rate (CBR; RECIST/GCIG CA-125 or therapy duration ≥16w), ctDNA molecular response, PK and PD. To evaluate lunresertib in combination with Debio 0123, a highly selective, brain-penetrant, clinical WEE1 inhibitor, in Module 4 of the ongoing MYTHIC trial in patients with advanced solid tumors harboring CCNE1 amplification or FBXW7 or PPP2R1A deleterious alterations. Dose expansion: To evaluate lunresertib in combination with camonsertib in patients with ovarian and endometrial cancers To evaluate dose expansion clinical trial of lunresertib and camonsertib at the recommended Phase 2 dose (RP2D) in patients with platinum-resistant ovarian and endometrial cancers harboring CCNE1 amplification or FBXW7 or PPP2R1A mutations To analyse factors associated with anaemia risk and implemented pt specific schedule and modification strategies (dose or schedule) to optimise tolerability. Exposure response (E-R) analysis assessed daily PK exposures at steady state (AUC and Cmax) vs. the probability of anaemia. Cox regression models assessed baseline (BL) predictive factors of Gr3 anaemia in pts at RP2D range. Overall rates and exposure-adjusted event rate (Gr3 events/pt-month of observation) of anaemia were analysed to assess the impact of the schedule optimisation approach. Anti-tumour activity was assessed radiographically (RECISTv1.1) and by exploratory circulating markers. Endpoints were response rate (RECISTv1.1 unconfirmed+confirmed), clinical benefit rate (CBR; response per RECISTv1.1 or treatment for ≥16w w/o PD), progression-free survival (PFS), and molecular response rate (MRR; ≥50% decline in circulating tumor DNA). Target and pathway engagement were evaluated by IHC in paired tumor biopsies.

Objectives included safety, tolerability, recommended doses (RD), pharmacokinetics (PK), pharmacodynamics (PDy), antitumor activity and molecular response rate (MRR; ≥50% decline in circulating tumor DNA).

Clinical Study Information for Healthcare Providers

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Study Locations

Location
Investigator
Status
Condition(s) Treated at Site
Location
START Midwest
Grand Rapids, MI, United States, 49546
Investigator
Nehal Lakhani
Status
Recruiting
Condition(s) Treated at Site
Solid Tumor